The Academy by Psych Scene

Psychiatry doesn’t slow down.

New research, updated guidelines, shifting frameworks, the field is constantly evolving. Keeping everything organised, current, and clinically useful in one place is almost impossible when you’re managing a full patient load and a personal life.

That’s where The Academy comes in.

Instead of scattered journals, fragmented CPD activities, and disconnected resources, you get one structured, clinician-focused platform.

Here’s what you get:

-150+ hours of expert-led, evidence-based content
-CME/CPD accreditation + free CPD Portfolio Tracker
-Flexible, on-demand video and audio learning
-Practical, clinician-ready frameworks
-Direct teaching from psychiatry experts
-Global peer collaboration
-Downloadable tools + instant certificates
-Full access for approximately AUD $1.64/day

Everything you need. One place.

Stay current. Stay confident. Future-proof your practice.

Join The Academy — click the link in the comments below:

21 hours ago | [YT] | 2

The Academy by Psych Scene

Why Are Substance Use Disorders (SUDs) Common in Borderline Personality Disorder (BPD) Patients?

Most assume that substance use in BPD is merely a secondary coping mechanism for acute emotional distress.

However, with a reported lifetime prevalence of 78%, clinical data suggest that specific neurocognitive deficits like high trait impulsivity and altered reward processing, are associated with this high comorbidity frequency.

Here is a breakdown of the mechanisms linking BPD to SUDs.

To learn more about the nuanced link between substance use disorders and Borderline Personality Disorder (BPD), click the link in the comments below and check out the full article on Psych Scene Hub.

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1 day ago | [YT] | 5

The Academy by Psych Scene

Can Prolonged Lithium Therapy Contribute to Chronic Kidney Disease (CKD)?

While lithium remains the gold standard for mood stabilisation, chronic exposure carries a cumulative risk of progressive renal dysfunction.

Existing clinical data indicate that prolonged lithium exposure can disrupt distal tubular homeostasis, leading to a specific tubulointerstitial pathology that may progress to irreversible CKD.

Here’s what clinicians need to know about the lithium-CKD link.

To learn more about the pathophysiology and management of lithium-induced Chronic Kidney Disease (CKD), click the link in the comments below and check out the full article on Psych Scene Hub.

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4 days ago | [YT] | 6

The Academy by Psych Scene

Metabolic management remains one of the major challenges in psychiatric practice.

Clinicians often come face-to-face with the clinical dilemma of treating mental illnesses, all while managing the significant metabolic risks associated with existing pharmacological interventions.

However, with the rise of GLP-1 receptor agonists and other metabolic therapies, new opportunities to strengthen one’s approach to metabolic management are now within reach.

Join leading endocrinologist Professor Roger Chen as he explores the role of GLP-1 receptor agonists and emerging agents in psychiatric care, particularly in addressing metabolic risks associated with antipsychotics.

Webinar Date: 24 February 2026 | Tuesday 7:00pm – 8:30pm AEDT
CPD: 1.5 CPD hours | Educational Activities

What We Cover In The Webinar:

How metabolic dysfunction influences psychiatric outcomes and recovery

Mechanisms of GLP-1 receptor agonists and which patients benefit most

Practical prescribing: selection, titration, side effects, and monitoring

Managing psychotropic-related weight gain, insulin resistance, and dysglycaemia

Emerging metabolic therapies relevant to mental health practice

For Academy Members: Free | includes exclusive on-demand access

For Non-Academy Members: $89 AUD | includes live attendance + 1 month on-demand viewing.

Reserve your spot now—link in the comments below.

1 week ago (edited) | [YT] | 9

The Academy by Psych Scene

Can Antipsychotics Induce Weight Gain?

Clinical data indicate that up to 72% of patients treated with second-generation antipsychotics experience weight gain.

While most dismiss it as lifestyle-induced, its pathology points to a drug-induced disruption of both the patient's central appetite control system and peripheral glucose metabolism.

Here's a clinical breakdown of how antipsychotics can induce weight gain.

To learn more about the pathophysiology of antipsychotic-induced weight gain, click the link in the comments below and check out the full article on Psych Scene Hub.

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1 week ago | [YT] | 9

The Academy by Psych Scene

ADHD diagnosis is often reduced to symptom checklists, yet real-world, clinical cases are rarely that simple.

Led by Dr Sanil Rege, our live ADHD Masterclass provides clinicians with a clear, clinically usable framework grounded in neurobiology and real, evidence-based cases, designed to help strengthen ADHD diagnostic accuracy and improve treatment precision.

In this course, you’ll learn how to:

Apply systems-based and developmental frameworks to ADHD presentations

Understand dopamine/noradrenaline network dynamics, arousal regulation, and network switching

Identify ADHD using a structured, multi-modal diagnostic framework (history, collateral, tools)

Distinguish comorbidity and drivers of partial/non-response using a staged diagnostic hierarchy

Integrate behavioural/lifestyle interventions alongside ADHD pharmacotherapy

Accreditation:

✅ 7.5 CPD hours (EA 6, RP 1.5)

✅ Self-accreditable with RANZCP

✅ RACGP & ACRRM accredited

✅ Recognised within GPMHSC FPS CPD frameworks

Start learning today. Link in the comment section.

1 week ago | [YT] | 8

The Academy by Psych Scene

Can Brain Insulin Resistance (BIR) Drive Alzheimer's Disease (AD)?

Most see metabolic disorders, such as BIR, and Alzheimer's as comorbid but distinct conditions.

However, clinical data suggest that BIR acts as a contributor to neurodegenerative processes seen in AD by disrupting central glucose metabolism and synaptic plasticity.

Here’s a neurobiological breakdown of the BIR-AD link.

To learn more about the nuanced neurobiological link between Brain Insulin Resistance (BIR) and Alzheimer's Disease (AD), click the link in the comments below and check out the full article on Psych Scene Hub.

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2 weeks ago | [YT] | 6

The Academy by Psych Scene

Why Does Standard Exposure Therapy Sometimes Underperform in Dissociative PTSD?

Standard PTSD interventions often focus on reducing hyperarousal. However, dissociative symptoms can occur in approximately 15–30% of patients with PTSD, and neuroimaging studies have described distinct patterns of emotion modulation in this subgroup (Lanius et al., 2006).

For these patients, clinicians should assess dissociation before initiating exposure-based treatment, as dissociation has been linked to poorer treatment outcomes and can be associated with symptom worsening when not recognised and managed appropriately.

Here’s a breakdown of how the dissociative phenotype can interfere with exposure-based approaches:🧵👇

To learn more about the intricacies of the dissociative PTSD phenotype and dive deeper into the neurobiological spectrum of trauma, click the link in the comments below and check out the full article on Psych Scene Hub.

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2 weeks ago | [YT] | 6

The Academy by Psych Scene

Can chronically elevated insulin levels contribute to brain insulin resistance (BIR)?

Chronic hyperinsulinaemia and BIR are often treated as separate metabolic phenomena.

However, evidence suggests elevated insulin levels are associated with impaired insulin transport across the blood–brain barrier, contributing to reduced central insulin signalling.

This peripheral–central mismatch is increasingly observed in metabolic and neurodegenerative disorders.

Here’s how this relationship unfolds 👇🧵

To learn more about the nuanced neurobiological link between chronically elevated insulin levels and Brain Insulin Resistance, click the link in the comments below and check out the full article on Psych Scene Hub.

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2 weeks ago | [YT] | 7

The Academy by Psych Scene

Why is Postpartum Psychosis (PPP) Often Misdiagnosed as Postnatal Depression (PND)?

At face value, it’s not surprising how most see PPP and PND as similar points on a spectrum of postnatal mood distress.

However, clinical data suggest that while both manifest affective symptoms, PPP is driven by a hyper-dopaminergic state as compared to PND's typically non-psychotic, slower-onset depressive pathology.

Here’s a neurobiological breakdown of this nuanced distinction between PPP and PND.

To learn more about the nuanced distinction between Postpartum Psychosis (PPP) and Postnatal Depression (PND), click the link below and check out the full article on Psych Scene Hub:

psychscene.co/45PFfih

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3 weeks ago | [YT] | 7